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Which class of medication protects the ischemic myocardium by blocking catecholamines and sympathetic nerve stimulation?

Which class of medication protects the ischemic myocardium by blocking catecholamines and sympathetic nerve stimulation?

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Asked by Sofia, Last updated: Nov 09, 2024

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J. Shatner

J. Shatner

J. Shatner
J. Shatner, Content writer, Boston

Answered Oct 15, 2018

Myocardial ischemia is a term referring to the lack or decrease of blood flowing to the heart. This causes the heart to not get enough oxygen that it should or is used to. After having myocardial ischemia, this could lead to heart problems, and even a blockage which could cause a heart attack. If you have this disease, then you may feel pain in your arm, shoulder, neck or jaw. You also will feel tired and sick to you stomach.

Catecholamines are produced in the body and are part of the amino acids in the body. The medication that protects the ischemic myocardium by blocking catecholamines and sympathetic nerve stimulation is beta adrenergic blockers. Otherwise, you risk the chance of having a heart attack.

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John Smith

John Smith

John Smith
John Smith

Answered Sep 09, 2016

Beta-adrenergic blockers-rationale: beta-adrenergic blockers work by blocking beta receptors in the myocardium, reducing the response to catecholamines and sympathetic nerve stimulation. they protect the myocardium, helping to reduce the risk of another infarction by decreasing myocardial oxygen demand. calcium channel blockers reduce the hearts workload by decreasing the heart rate. opioids reduce myocardial oxygen demand, promote vasodilation, and decrease anxiety. nitrates reduce myocardial oxygen consumption by decreasing left ventricular end-diastolic pressure (preload) and systemic vascular resistance (afterload).client needs category: physiological integrityclient needs subcategory: pharmacological and parenteral therapiescognitive level: applicationreference: abrams, a.c., et al. clinical drug therapy: rationales for nursing practice, 8th ed. lippincott williams & wilkins, 2007, p. 296.
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